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The HMB, which stands for hydroxymethylbutyrate, is the main metabolite in the branched-chain amino acid leucine that prevents the breakdown of muscle proteinin the process of muscle fatigue following resistance training. An increase in endogenous leucine levels leads to the creation of free leucine as a precursor to muscle protein breakdown. "Lec-1 is an important regulator of the production of l-[1,2-c]leucine, which then facilitates the uptake of muscle protein from the intestine into the muscle," said López-Ruiz. The team set out to test whether exercise training would reduce the production of endogenous leucine in healthy young males. Twenty-four young male subjects (13.7 +/- 2.2 years) were randomly divided into both the placebo and exercise group. A 5-week training period followed. They undertook resistance training, with three sets at 70% of the subjects' 1RM strength at 90% of the estimated 1RM, twice daily for six weeks. The researchers measured the production of leucine, which was measured using a novel metabolic marker, r-rho. The authors were able to conclude that when compared with placebo, exercise increased the plasma concentrations of r-rho. The group performing the exercise also exhibited decreased r-rho levels in response to exercise training. This study demonstrates r-rho is a marker of endogenous leucine synthesis in vivo. We now plan to examine whether a similar result can be obtained under free feeding conditions. In this context, we recommend that the authors develop a placebo group in this study so that the effect of exercise cannot be evaluated with a dietary supplement. "We found that exercise increased the endogenous levels of leucine, and this effect was more pronounced following training," said López-Ruiz. "A similar increase in leucine has been reported after several weeks of intense exercise training in athletes. This could mean that the increase found would only last for a period of only a couple of days after the exercise training exercise session." The study was funded by the Natural Sciences and Engineering Research Council of Canada, and the Royal Society of Canada, and received funding support from the Canadian Institutes of Health Research (Fonds de Recherche en Santé et de Recherche sur l'Épidémiologie et du développement National de l'Avenir). Article: "Reciprocal effect of exercise and free feeding on serum levels of r-rho and serum insulin-like growth factor 1" by López-Ruiz, Similar articles:

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